‘The Elixir of Youth’? Omega-3s linked to younger

Telomeres, DNA sequences at the stop of chromosomes that shorten as cells replicate and age, does not normally correlate with chronological age, and there is proof to suggest telomere shortening may perhaps be modifiable by lifestyle variables.

“The factors that are strongly involved with accelerated telomere shortening and dysfunction are oxidative strain and swelling,” ​explained scientists from Institute of Genetics and Animal Biotechnology of the Polish Academy of Sciences in Nutrients​.

“The skill of omega‐3 fatty acids to reduce these adverse effects is relevant not only to their well‐documented effective influence on a selection of ‘lifestyle’ illnesses but also to their useful effects on telomere biology.

“The use of omega‐3 fatty acids to reduce accelerated telomere attrition and, for that reason, counteract untimely growing older and minimize the hazard of age‐related health conditions raises high hopes.”


The growing old and lifespan of usual, healthful cells are joined to the so-called telomerase shortening mechanism, which limitations cells to a set amount of divisions. In the course of cell replication, the telomeres perform by making sure the cell’s chromosomes do not fuse with each other or rearrange, which can guide to most cancers.

Elizabeth Blackburn, a telomere pioneer at the University of California San Francisco, likened telomeres to the ends of shoelaces, without which the lace would unravel.

With each and every replication the telomeres shorten, and when the telomeres are entirely consumed, the cells are wrecked (apoptosis). Telomere shortening or attrition was shown as a person of the nine hallmarks of growing old in a seminal paper published in Cell​ in 2013 by Carlos López-Otín et al​.

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